Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the flip side, our power to rescue SERT function in KI midbrain synaptoneurosomes from the inhibition of FAK indicates elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by amplified pFAK localization in https://pro3388888.bloggerswise.com/39404496/an-unbiased-view-of-pro33-login