Multiple mitochondrial defects ended up present in the mouse product of Pompe disease, which includes mitochondrial calcium surplus, enhanced reactive oxygen species, reduced mitochondrial membrane possible, and lowered oxygen consumption and ATP manufacturing (Lim et al (2015) A murine Niemann‐Pick C1 I1061T knock‐in design recapitulates the pathological capabilities of by https://shigesatog432sfs6.mybuzzblog.com/profile